Most people can only dream of a life without pain or anxiety, but for a 71-year-old woman in Scotland, this dream is their reality.
Jo Cameron recently found out she has a rare genetic mutation that prevents her from experiencing pain, anxiety or anxiety. It can also cause her wounds to heal faster and make them forget.
An article on Cameron's condition was published Thursday in the British Journal of Anesthesia. According to study authors from the University College of London, their conditions may help scientists develop new therapies for multiple diseases.
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Throughout her life, Cameron had scratches and burns and cuts, but had not felt pain and often only noticed burns until, according to the publication burning meat smelled. She did not need any painkillers after surgery and told the BBC that she did not feel any pain during childbirth – even without an epidural. "
" It was just weird, but I was in no pain. It was really nice. "She told the socket.
It was only when she was 65 and had hip problems – which turned out to be a severe joint degeneration – that she realized how startling her painlessness really was.
After The Surgery The doctors saw that she needed a two-handed operation that did not require painkillers, The Guardian reported.
One of her doctors, dr. Devjit Srivastava, consultant for anesthesia and pain medicine, referred her to pain specialists at University College London and the University of Oxford.
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Mutations in genes that relate to pain, mood, and memory. It used to be thought that one of these genes has no function.
Now, scientists believe that these mutations prevent Cameron from feeling pain and anxiety, causing them to heal faster, be more positive and forgetful.
In their analysis, the researchers also found that Cameron was rated low on an anxiety score. She told them that in dangerous situations she did not panic and had memory lapses in her life and sometimes forgot words or objects like her keys.
"We found that this woman has a specific genotype that reduces the activity of an already-considered gene James Cox, from UCL Medicine and one of the study's leading researchers, said in the publication.
"Now that we know how this newly identified gene works, we hope to make further progress in new treatment goals," Cox added.